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Glyceryl Trioleate

$364

  • Brand : BIOFRON

  • Catalogue Number : BD-P0970

  • Specification : 100%

  • CAS number : 122-32-7

  • Formula : C57H104O6

  • Molecular Weight : 885.44

  • PUBCHEM ID : 5497163

  • Volume : 0.15ml

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Catalogue Number

BD-P0970

Analysis Method

Specification

100%

Storage

-20℃

Molecular Weight

885.44

Appearance

Oil

Botanical Source

This product is isolated and purified from the leaves of Canarium album

Structure Type

Category

SMILES

CCCCCCCCC=CCCCCCCCC(=O)OCC(COC(=O)CCCCCCCC=CCCCCCCCC)OC(=O)CCCCCCCC=CCCCCCCCC

Synonyms

Glycerin trioleate/Olein 9 C18:1 cis/Glycerol triolein/Triolein/glyceryl trioleate/Glycerine trioleate/tri-olei/(9Z)9-Octadecenoic acid 1,2,3-propanetriyl ester (1,2,3-Tri(cis-9-octadecenoyl)glycerol/9-Octadecenoic acid (9Z)-, 1,2,3-propanetriyl ester/1,2,3-tri-(9Z-octadecenoyl)-sn-glycerol/raoline/OLEIN/9-Octadecenoic-9,10-t2 acid, 1,2,3-propanetriyl ester, (Z,Z,Z)- (9CI)/9-Octadecenoic acid, 1,2,3-propanetriyl ester, (9Z,9'Z,9''Z)/Oleic acid triglyceride/(9Z)9-Octadecenoic acid 1,2,3-propanetriyl ester/aldoto/9-Octadecenoic acid (Z)-, 1,2,3-propanetriyl ester/18:1TG/Glycerol, tri(cis-9-octadecenoate)/Glyceryl-1,2,3-trioleate/Oleic triglyceride/1,2,3-Tri(cis-9-octadecenoyl)glycerol/Olein, tri-/Propane-1,2,3-triyl (9Z,9'Z,9''Z)tris-octadec-9-enoate/Radia 7363/Glycerol trioleate/1,2,3-tri(cis-9-octadecenoyloxy)propane/1,2,3-Propanetriyl (9Z,9'Z,9''Z)tris(-9-octadecenoate/emery2423/Erdnuoel/Trioleoylglyceride

IUPAC Name

Applications

Density

0.9±0.1 g/cm3

Solubility

Flash Point

302.7±31.5 °C

Boiling Point

818.7±55.0 °C at 760 mmHg

Melting Point

-5,5°C

InChl

InChl Key

PHYFQTYBJUILEZ-IUPFWZBJSA-N

WGK Germany

RID/ADR

HS Code Reference

Personal Projective Equipment

Correct Usage

For Reference Standard and R&D, Not for Human Use Directly.

Meta Tag

provides coniferyl ferulate(CAS#:122-32-7) MSDS, density, melting point, boiling point, structure, formula, molecular weight etc. Articles of coniferyl ferulate are included as well.>> amp version: coniferyl ferulate

No Technical Documents Available For This Product.

PMID

31178460

Abstract

Camellia oleifera, C. japonica and C. sinensis are three representative crops of the genus Camellia. In this work, we systematically investigated the lipid characteristics of these seed oils collected from different regions. The results indicated significant differences in acid value (AV), peroxide value (PV), iodine value (IV), saponification value (SV) and relative density of the above-mentioned camellia seed oils (p < 0.05). The C. japonica seed oils showed the highest AV (1.7 mg/g), and the C. sinensis seed oils showed the highest PV (17.4 meq/kg). The C. japonica seed oils showed the lowest IV (79.9 g/100 g), SV (192.7 mg/g) and refractive index (1.4633) of all the oils, while the C. sinensis seed oils showed the lowest relative density (0.911 g/cm3). The major fatty acids in the camellia seed oils were palmitic acid (16:0), oleic acid (18:1) and linoleic acid (18:2); the oleic acid in C. oleifera and C. japonica seed oils accounted for more than 80% of the total fatty acids. The oleic acid levels in the C. oleifera and C. japonica oils were higher than those in the C. sinensis seed oils, while the linoleic acid levels in the former were lower than those in the latter one. Differences also exist in the triacylglycerol (TAG) composition, although the most abundant TAG molecular species in the camellia seed oils was trioleoylglycerol (OOO). Seven sterol species, squalene and α-tocopherol were detected in the camellia seed oils, however, the contents of tocopherol and unsaponifiable molecules in the C. oleifera and C. japonica seed oils were significantly lower than those in the C. sinensis seed oil. These results demonstrated that the varieties of Camellia affected the seed oil lipid characteristics.

KEYWORDS

camellia seed oil; physicochemical properties; systematic comparison

Title

Lipid Characteristics of Camellia Seed Oil.

Author

Zeng W1, Endo Y1.

Publish date

2019 Jul 1

PMID

30801996

Abstract

This protocol provides a comprehensive reference for the evolution of the lymph fistula model, the mechanism of lipid absorption, the detailed procedure for studying lipid absorption using the lymph fistula model, the interpretation of the results, and consideration of the experimental design. The lymph fistula model is an approach to assess the concentration and rate of a range of molecules transported by the lymph by cannulating lymph duct in animals. In this protocol, mice first undergo surgery with the implantation of cannulae in the duodenum and mesenteric lymph duct and are allowed to recover overnight in Bollman restraining cages housed in a temperature-regulated environment. To study in vivo lipid absorption, a lipid emulsion is prepared with labeled tracers, including [3 H]-triolein and [14 C]-cholesterol. On the day of the experiment, mice are continuously infused with lipid emulsion via the duodenum for 6 hr, and lymph is usually collected hourly. At the end of the study, gastrointestinal segments and their luminal contents are collected separately for determination of the digestion, uptake, and transport of exogenous lipids.

© 2019 by John Wiley & Sons, Inc

KEYWORDS

gastrointestinal tract; intestinal lymph duct; lipid absorption; lymph fistula

Title

Use of Isotope Tracers to Assess Lipid Absorption in Conscious Lymph Fistula Mice.

Author

Ko CW1, Qu J1, Liu M1, Black DD2, Tso P1.

Publish date

2019 Mar

Abstract

The transcription factor, PPARδ is involved in suppressing inflammation, stimulating oligodendroglial biogenesis and myelination. Furthermore, activation of PPARδ directly protects mitochondria against noxious stimuli and stimulates biogenesis of new mitochondria. PPARδ activation directly inhibits neuronal cell death and reduces both the level and neurotoxicity of Amyloid-β fibers in Alzheimer’s Disease (AD) models. Among the important ligands of PPARδ is erucic acid (EA, 22:1 n9), an edible omega-9 fatty acid and a component of Lorenzo’s oil, which is used in the treatment of adrenoleukodystrophy (ALD). Nonetheless, the feature of PPARδ-erucic acid interaction has not been extensively studied. EA can also be converted to nervonic acid, an important component of myelin. Hence, EA may act as an anti-inflammatory and remyelinating agent, which might be important in the management of another demyelinating disease, multiple sclerosis (MS). Oxidative injury and mitochondrial damage are among the features of ALD. Direct inhibitory effects of EA was observed on lipid peroxidation and inflammatory enzymes, neutrophil elastase and thrombin. EA also induces catalase, a potent antioxidant peroxisomal enzyme. However, EA is claimed to be a cardiotoxic molecule, yet these studies were mostly performed on rats, which do not efficiently metabolize EA. Further, EA is largely consumed by Asian population and Greenland Eskimos with no signs of cardiac damage. In this review, we shed light on the potential theraputic role of EA in MS and AD by blocking neural cell death, mitigating neuroinflammation and/or inducing myelination.

Copyright © 2019 Elsevier B.V. All rights reserved.

KEYWORDS

Alzheimer's Disease; Erucic acid; Multiple sclerosis; Neuroprotection; PPAR-delta; Remyelination

Title

PPAR-δ and erucic acid in multiple sclerosis and Alzheimer's Disease. Likely benefits in terms of immunity and metabolism.

Author

Altinoz MA1, Ozpinar A2.

Publish date

2019 Apr