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Resveratrol

$43

  • Brand : BIOFRON

  • Catalogue Number : BF-R2001

  • Specification : 98%

  • CAS number : 501-36-0

  • Formula : C14H12O3

  • Molecular Weight : 228.24

  • PUBCHEM ID : 445154

  • Volume : 20mg

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Catalogue Number

BF-R2001

Analysis Method

HPLC,NMR,MS

Specification

98%

Storage

-20℃

Molecular Weight

228.24

Appearance

White crystalline powder

Botanical Source

Vitis vinifera,Morus alba,Paeonia suffruticosa,Dracaena cochinchinensis,Reynoutria japonica

Structure Type

Stilbenes

Category

Standards;Natural Pytochemical;API

SMILES

C1=CC(=CC=C1C=CC2=CC(=CC(=C2)O)O)O

Synonyms

5-[(E)-2-(4-Hydroxyphenyl)ethenyl]benzol-1,3-diol/1,3-Benzenediol, 5-[(E)-2-(4-hydroxyphenyl)ethenyl]-/RESVERTROL/3',4,5'-trihydroxy-trans-stilbene/1,3-Benzenediol, 5-(2-(4-hydroxyphenyl)ethenyl)-, (E)-/3,4',5-Trihydroxy-trans-stilbene/(E)-5-(p-Hydroxystyryl)resorcinol/trans-3,5,4'-trihydroxy-stilbene/5-[(1E)-2-(4-Hydroxyphenyl)ethenyl]-1,3-benzenediol/(E)-5-[2-(4-hydroxyphenyl)ethenyl]-1,3-benzendiol/3,5,4'-Trihydroxy-trans-stilbene/5-[(E)-2-(4-Hydroxyphenyl)vinyl]benzene-1,3-diol/RESVERATROLE/3,4',5-trihydroxy-stilbene/Resveratrol/TRANS-RESVERATROL/5-[(E)-2-(4-Hydroxyphenyl)vinyl]-1,3-benzenediol/5-[(E)-2-(4-hydroxyphenyl)ethenyl]benzene-1,3-diol/trans-1,2-(3,4',5-Trihydroxydiphenyl)ethylene/(E)-5-(4-hydroxystyryl)benzene-1,3-diol/5-[2-(4-hydroxyphenyl)ethenyl]benzene-1,3-diol

IUPAC Name

5-[(E)-2-(4-hydroxyphenyl)ethenyl]benzene-1,3-diol

Density

1.4±0.1 g/cm3

Solubility

Methanol

Flash Point

222.3±14.7 °C

Boiling Point

449.1±14.0 °C at 760 mmHg

Melting Point

253-255°C

InChl

InChl Key

WGK Germany

RID/ADR

HS Code Reference

2907290000

Personal Projective Equipment

Correct Usage

For Reference Standard and R&D, Not for Human Use Directly.

Meta Tag

provides coniferyl ferulate(CAS#:501-36-0) MSDS, density, melting point, boiling point, structure, formula, molecular weight etc. Articles of coniferyl ferulate are included as well.>> amp version: coniferyl ferulate

PMID

30000939

Title

Resveratrol.

Publish date

2006

PMID

21175036

Abstract

The present study was designed to examine the potential preventive effect of curcumin (CMN; CAS 458-37-7), rosiglitazone (RGN; CAS 155141-29-0), N-acetylcysteine (NAC; CAS 616-91-1), resveratrol (RSV; CAS 501-36-0), and losartan (LOS; CAS 114798-26-4) on sodium valproate-induced hepatotoxicity. Sodium valproate (SVP; CAS 1069-66-5) was given at a dose of 250 mg/kg i. p. 3 times daily for one week. The tested compounds were given simultaneously with SVP for one week. The results demonstrate that CMN, RGN and NAC treatment can confer protection from SVP-induced hepatotoxicity. The second part of the study includes an evaluation of the effect of CMN, RGN and NAC on the anticonvulsant activity of SVP against pentetrazole-induced seizures in mice. The results demonstrate that CMN, RGN and NAC do not affect the anticonvulsant activity of SVP. Combined administration of either of CMN, RGN and NAC with valproate appears to be beneficial in reducing valproate-induced hepatotoxicity.

Title

Prevention of sodium valproate-induced hepatotoxicity by curcumin, rosiglitazone and N-acetylcysteine in rats.

Author

Said SA1, El-Agamy DS.

Publish date

2010

PMID

17225566

Abstract

Resveratrol (trans-3,4′,5-trihydroxystilbene, CAS 501-36-0), a natural antioxidant and polyphenol found in grapes and wine, has been found to pharmacologically precondition the heart in nitric oxide (NO)-dependent manner. In the vascular system, NO functions as an endogenous inhibitor of leukocyte chemotaxis, adherence, and activation. The present study was designed to determine if resveratrol, through NO, can block the proadhesive molecules generated in the ischemic reperfused myocardium. Isolated hearts were prepared from properly anesthetized rats, and mounted on a Langendorff apparatus. The hearts were randomly assigned to one of the three groups: (i) control, (ii) resveratrol, and (iii) resveratrol + NG-nitro-L-arginine ethyl ester (L-NAME). The hearts were perfused in the absence (n = 6) or presence of 10 micromol/L resveratrol (n=6) or resveratrol + L-NAME (n = 6) for 15 min. All the hearts were then subjected to 30 min ischemia followed by 2 h of reperfusion. Ventricular function was monitored, infarct size and apoptotic cell death measured, and the proadhesive molecules and malonaldehyde formation determined in the perfusate. Resveratrol significantly improved postischemic ventricular function and reduced myocardial infarct size compared to the non-treated control group. The amount of proadhesive molecules including soluble intracellular adhesion molecule-1 (sICAM-1), endothelial leukocyte adhesion molecule-1 (sE-Selectin) and vascular cell adhesion molecule-1 (sVCAM-1) were each significantly decreased during reperfusion in the resveratrol group. L-NAME, a NO blocker, completely abolished such beneficial effects of resveratrol. The results support an anti-inflammatory action of resveratrol through a NO-dependent mechanism.

Title

Attenuation of ischemia/reperfusion injury in rats by the anti-inflammatory action of resveratrol.

Author

Das S1, Falchi M, Bertelli A, Maulik N, Das DK.

Publish date

2006


Description :

Resveratrol (SRT 501), a natural polyphenol that possesses anti-oxidant, anti-inflammatory, cardioprotective, and anti-cancer properties. It has a wide spectrum of targets including mTOR, JAK, β-amyloid.